The paradoxical role of thioredoxin on oxidative stress and aging
Geneva M. Cunningham(Indiana University – Purdue University Indianapolis), Yuji Ikeno(The University of Texas at San Antonio Health Science Center), Lisa C. Flores, Madeline G. Roman(The University of Texas Health Science Center at San Antonio), Yiqiang Zhang(Heilongjiang University of Chinese Medicine), Jonathan Gelfond(The University of Texas at San Antonio Health Science Center), Gene B. Hubbard(The University of Texas at San Antonio Health Science Center), Adam B. Salmon(The University of Texas at San Antonio Health Science Center)
Cited by 61
Related Papers
Life-long reduction in MnSOD activity results in increased DNA damage and higher incidence of cancer but does not accelerate aging
|Physiological Genomics|2003|733
Is the oxidative stress theory of aging dead?
|Biochimica et Biophysica Acta (BBA) - General Subjects|2009|602
Obesity-Induced Cellular Senescence Drives Anxiety and Impairs Neurogenesis
|Cell Metabolism|2019|547
Insulin resistance is a cellular antioxidant defense mechanism
|Proceedings of the National Academy of Sciences|2009|541
Rapamycin Extends Life and Health in C57BL/6 Mice
|The Journals of Gerontology Series A|2013|284