Dose-Dependent AMPK-Dependent and Independent Mechanisms of Berberine and Metformin Inhibition of mTORC1, ERK, DNA Synthesis and Proliferation in Pancreatic Cancer Cells
Ming Ming(University of California, Los Angeles), Enrique Rozengurt(University of California, Los Angeles)
Cited by 71
Related Papers
Activation Loop Ser744 and Ser748 in Protein Kinase D Are Transphosphorylated in Vivo
|Journal of Biological Chemistry|2001|158
Different Patterns of Akt and ERK Feedback Activation in Response to Rapamycin, Active-Site mTOR Inhibitors and Metformin in Pancreatic Cancer Cells
|PLoS ONE|2013|129
Protein Kinase D Potentiates DNA Synthesis and Cell Proliferation Induced by Bombesin, Vasopressin, or Phorbol Esters in Swiss 3T3 Cells
|Journal of Biological Chemistry|2001|99
Protein Kinase Cν/Protein Kinase D3 Nuclear Localization, Catalytic Activation, and Intracellular Redistribution in Response to G Protein-coupled Receptor Agonists
|Journal of Biological Chemistry|2003|92
Sequential Protein Kinase C (PKC)-dependent and PKC-independent Protein Kinase D Catalytic Activation via Gq-coupled Receptors
|Journal of Biological Chemistry|2008|86