The β1 subunit of Na<sup>+</sup>/K<sup>+</sup>‐ATPase interacts with BK<sub>Ca</sub> channels and affects their steady‐state expression on the cell surface

Smita Jha(National Institutes of Health), Stuart E. Dryer(University of Houston)

FEBS Letters

September 1, 2009

10.1016/j.febslet.2009.08.039

Cited by 17

Related Papers

Sustained Activation of N-Methyl-d-Aspartate Receptors in Podoctyes Leads to Oxidative Stress, Mobilization of Transient Receptor Potential Canonical 6 Channels, Nuclear Factor of Activated T Cells Activation, and Apoptotic Cell Death

|Molecular Pharmacology|2012|52

Retrospective study of inpatient diabetes management service, length of stay and 30-day readmission rate of patients with diabetes at a community hospital

|Journal of Community Hospital Internal Medicine Perspectives|2019|44

Dominant-Negative Regulation of Cell Surface Expression by a Pentapeptide Motif at the Extreme COOH Terminus of an Slo1 Calcium-Activated Potassium Channel Splice Variant

|Molecular Pharmacology|2010|33

<scp>TRPC6</scp> effects on albumin permeation, nephrin shedding, and apoptosis in podocytes: Role of calcineurin and metalloproteases

|Physiological Reports|2025|0