Towards a therapy for Angelman syndrome by targeting a long non-coding RNALinyan Meng(Texas Children's Hospital), Amanda J. Ward, Seung Chun, C. Frank Bennett, Arthur L. Beaudet(Texas Children's Hospital), Frank RigoNatureDecember 1, 201410.1038/nature13975Cited by 524SaveCiteExport RISWatch citationsAbstractRelated PapersPrader-Willi syndromeSuzanne B. Cassidy, Stuart Schwartz, Jennifer Miller et al.|Genetics in Medicine|2012|1.4kUBE3A/E6-AP mutations cause Angelman syndromeTatsuya Kishino, Marc Lalande, Joseph Wagstaff|Nature Genetics|1997|1.3kDe novo truncating mutations in E6-AP ubiquitin-protein ligase gene (UBE3A) in Angelman syndromeToshinobu Matsuura, James S. Sutcliffe, Ping Fang et al.|Nature Genetics|1997|875Mutation of the Angelman Ubiquitin Ligase in Mice Causes Increased Cytoplasmic p53 and Deficits of Contextual Learning and Long-Term PotentiationYong‐hui Jiang, Dawna Armstrong, Urs Albrecht et al.|Neuron|1998|867Sustained Therapeutic Reversal of Huntington's Disease by Transient Repression of Huntingtin SynthesisHolly Kordasiewicz, Lisa M. Stanek, Edward V. Wancewicz et al.|Neuron|2012|765