Genetic and Infectious Profiles of Japanese Multiple Sclerosis Patients
Satoshi Yoshimura(St. Jude Children's Research Hospital), Jun‐ichi Kira(Kyushu University), Tomomi Yonekawa(Kyushu University), Takuya Matsushita(Kyushu University), Ken Yamamoto, Shinya Sato(Kyushu University), Noriko Isobe(Kyushu University), Yuji Kawano(Ehime University), Katsuhisa Masaki(University of Chicago Medical Center)
Cited by 78
Related Papers
Involvement of a NF-kappa B-like transcription factor in the activation of the interleukin-6 gene by inflammatory lymphokines.
|Molecular and Cellular Biology|1990|378
Genome-wide association study of clinically defined gout identifies multiple risk loci and its association with clinical subtypes
|Annals of the Rheumatic Diseases|2015|183
GWAS of clinically defined gout and subtypes identifies multiple susceptibility loci that include urate transporter genes
|Annals of the Rheumatic Diseases|2016|173
ABCG2 dysfunction causes hyperuricemia due to both renal urate underexcretion and renal urate overload
|Scientific Reports|2014|163
Distinct genetic and infectious profiles in Japanese neuromyelitis optica patients according to anti-aquaporin 4 antibody status
|Journal of Neurology Neurosurgery & Psychiatry|2012|134