The Potent Oncogene NPM-ALK Mediates Malignant Transformation of Normal Human CD4+ T Lymphocytes

Qian Zhang(University of Pennsylvania), Mariusz A. Wasik(Fox Chase Cancer Center)

American Journal Of Pathology

October 24, 2013

10.1016/j.ajpath.2013.08.030

Cited by 37

Related Papers

Oncogenic kinase NPM/ALK induces through STAT3 expression of immunosuppressive protein CD274 (PD-L1, B7-H1)

|Proceedings of the National Academy of Sciences|2008|723

Lack of Phosphotyrosine Phosphatase SHP-1 Expression in Malignant T-Cell Lymphoma Cells Results from Methylation of the SHP-1 Promoter

|American Journal Of Pathology|2000|128

IL-2– and IL-15–induced activation of the rapamycin-sensitive mTORC1 pathway in malignant CD4+ T lymphocytes

|Blood|2007|74

Activation of Mammalian Target of Rapamycin in Transformed B Lymphocytes Is Nutrient Dependent but Independent of Akt, Mitogen-Activated Protein Kinase/Extracellular Signal-Regulated Kinase Kinase, Insulin Growth Factor-I, and Serum

|Cancer Research|2005|72

Expression and Oncogenic Role of Brk (PTK6/Sik) Protein Tyrosine Kinase in Lymphocytes

|American Journal Of Pathology|2006|53