IL-18 induces the differentiation of Th1 or Th2 cells depending upon cytokine milieu and genetic background

Damo Xu(Wellcome Centre for Molecular Parasitology), Foo Y. Liew(University of Glasgow), Kerstin Schulz(Queen Mary University of London), Vladimir Trajković(University of Belgrade), J. Alastair Gracie(University of Strathclyde), David J. Hunter(University of Glasgow), Iain B. McInnes(University of Glasgow), Bernard P. Leung
European Journal of Immunology
November 1, 2000
10.1002/1521-4141(200011)30:11<3147::aid-immu3147>3.0.co;2-j
Cited by 154

Related Papers

EULAR recommendations for the management of rheumatoid arthritis with synthetic and biological disease-modifying antirheumatic drugs: 2019 update
|Annals of the Rheumatic Diseases|2020|2.8k
EULAR recommendations for the management of rheumatoid arthritis with synthetic and biological disease-modifying antirheumatic drugs: 2022 update
|Annals of the Rheumatic Diseases|2022|1.9k
Incidence, prevalence, and co-occurrence of autoimmune disorders over time and by age, sex, and socioeconomic status: a population-based cohort study of 22 million individuals in the UK
|The Lancet|2023|838
A proinflammatory role for IL-18 in rheumatoid arthritis
|Journal of Clinical Investigation|1999|648
Interleukin-18
|Journal of Leukocyte Biology|2003|600