Substance P Induces CCN1 Expression via Histone Deacetylase Activity in Human Colonic Epithelial Cells
Hon Wai Koon, Charalabos Pothoulakis(University of California, Los Angeles)
Cited by 16
Related Papers
A long noncoding RNA signature for ulcerative colitis identifies IFNG-AS1 as an enhancer of inflammation
|American Journal of Physiology-Gastrointestinal and Liver Physiology|2016|120
Substance P Stimulates Cyclooxygenase-2 and Prostaglandin E2 Expression through JAK-STAT Activation in Human Colonic Epithelial Cells
|The Journal of Immunology|2006|102
CRHR2/Ucn2 signaling is a novel regulator of miR‐7/YY1/Fas circuitry contributing to reversal of colorectal cancer cell resistance to Fas‐mediated apoptosis
|International Journal of Cancer|2017|44
Substance P-Mediated Expression of the Pro-Angiogenic Factor CCN1 Modulates the Course of Colitis
|American Journal Of Pathology|2008|40
The Neurotensin–HIF-1α–VEGFα Axis Orchestrates Hypoxia, Colonic Inflammation, and Intestinal Angiogenesis
|American Journal Of Pathology|2014|34