Oxidative Stress Mediates Chemical Hypoxia-Induced Injury and Inflammation by Activating NF-κb-COX-2 Pathway in HaCaT Cells
Chuntao Yang(Kunming University of Science and Technology), Jianqiang Feng(Sun Yat-sen University)
Cited by 64
Related Papers
Activation of the p38 MAPK/NF-κB pathway contributes to doxorubicin-induced inflammation and cytotoxicity in H9c2 cardiac cells
|Molecular Medicine Reports|2013|139
Exogenous Hydrogen Sulfide Protects against Doxorubicin-Induced Inflammation and Cytotoxicity by Inhibiting p38MAPK/NFκB Pathway in H9c2 Cardiac Cells
|Cellular Physiology and Biochemistry|2013|101
Hydrogen sulfide attenuates doxorubicin-induced cardiotoxicity by inhibition of the p38 MAPK pathway in H9c2 cells
|International Journal of Molecular Medicine|2013|80
Exogenous hydrogen sulfide protects H9c2 cardiac cells against high glucose-induced injury by inhibiting the activities of the p38 MAPK and ERK1/2 pathways
|International Journal of Molecular Medicine|2013|61
Data‐Driven Identification of Hydrogen Sulfide Scavengers
|Angewandte Chemie International Edition|2019|49