Neprilysin-sensitive Synapse-associated Amyloid-β Peptide Oligomers Impair Neuronal Plasticity and Cognitive Function
Shu-Ming Huang(RIKEN Center for Brain Science), Nobuhisa Iwata(University of Nagasaki), Takaomi C. Saido(RIKEN Center for Brain Science), Akihiro Mouri(Fujita Health University), Matthias Staufenbiel(Novartis (Switzerland)), Toshitaka Nabeshima(Meijo University), Makoto Higuchi(National Institutes for Quantum Science and Technology), Takahiro Suemoto(RIKEN Center for Brain Science), Ryuichi Nakajima(RIKEN Center for Brain Science), Haruyasu Yamaguchi(Goethe University Frankfurt), Hideko Kokubo(Gunma University), Yukihiro Noda(Meijo University)
Cited by 177
Related Papers
<scp>APP</scp> mouse models for Alzheimer's disease preclinical studies
|The EMBO Journal|2017|780
Brain homogenates from human tauopathies induce tau inclusions in mouse brain
|Proceedings of the National Academy of Sciences|2013|761
Destabilization of β-catenin by mutations in presenilin-1 potentiates neuronal apoptosis
|Nature|1998|517
Two Types of Sporadic Cerebral Amyloid Angiopathy
|Journal of Neuropathology & Experimental Neurology|2002|450