Caspase‐3‐mediated cleavage of Akt: Involvement of non‐consensus sites and influence of phosphorylation
Arezu Jahani‐Asl(University of Ottawa), Benjamin K. Tsang(University of Ottawa)
Cited by 25
Related Papers
Akt promotes cisplatin resistance in human ovarian cancer cells through inhibition of p53 phosphorylation and nuclear function
|International Journal of Cancer|2007|185
Control of glioblastoma tumorigenesis by feed-forward cytokine signaling
|Nature Neuroscience|2016|110
Cisplatin Induces p53-Dependent FLICE-Like Inhibitory Protein Ubiquitination in Ovarian Cancer Cells
|Cancer Research|2008|85
P73 regulates cisplatin-induced apoptosis in ovarian cancer cells via a calcium/calpain-dependent mechanism
|Oncogene|2011|84
Cisplatin alters nitric oxide synthase levels in human ovarian cancer cells: involvement in p53 regulation and cisplatin resistance
|British Journal of Cancer|2008|74