Oxidative stress-induced overexpression of miR-25: the mechanism underlying the degeneration of melanocytes in vitiligo
Qiong Shi(Hebei Medical University), Chunying Li(Air Force Medical University)
Cited by 116
Related Papers
Signal pathways of melanoma and targeted therapy
|Signal Transduction and Targeted Therapy|2021|363
Nanoparticle delivery of miR-21-3p sensitizes melanoma to anti-PD-1 immunotherapy by promoting ferroptosis
|Journal for ImmunoTherapy of Cancer|2022|121
Dysregulated autophagy increased melanocyte sensitivity to H2O2-induced oxidative stress in vitiligo
|Scientific Reports|2017|116
Down-regulated miR-23a Contributes to the Metastasis of Cutaneous Melanoma by Promoting Autophagy
|Theranostics|2017|98
Activating Transcription Factor 4 (ATF4)-ATF3-C/EBP Homologous Protein (CHOP) Cascade Shows an Essential Role in the ER Stress-Induced Sensitization of Tetrachlorobenzoquinone-Challenged PC12 Cells to ROS-Mediated Apoptosis via Death Receptor 5 (DR5) Signaling
|Chemical Research in Toxicology|2016|61