Loss of allelic heterozygosity at a second locus on chromosome 11 in sporadic Wilms' tumor cells.
A E Reeve(University of Otago), Andrew P. Feinberg(Johns Hopkins University), S A Sih(The University of Texas MD Anderson Cancer Center), Anthony Raizis(Howard Hughes Medical Institute)
Cited by 205
Related Papers
Epigenome-wide association data implicate DNA methylation as an intermediary of genetic risk in rheumatoid arthritis
|Nature Biotechnology|2013|1k
Relaxation of insulin-like growth factor II gene imprinting implicated in Wilms' tumour
|Nature|1993|709
Genetic linkage of Beckwith-Wiedemann syndrome to 11p15.
|PubMed|1989|308
Constitutional relaxation of insulin–like growth factor II gene imprinting associated with Wilms' tumour and gigantism
|Nature Genetics|1993|114
Interferon-alpha restores the deficient expression of the cytoadhesion molecule lymphocyte function antigen-3 by chronic myelogenous leukemia progenitor cells.
|Journal of Clinical Investigation|1991|87