Role of gelsolin in the actin filament regulation of cardiac L-type calcium channels
Alan S. Lader(Harvard University), Horacio F. Cantiello(National University of Santiago del Estero), David J. Kwiatkowski(Brigham and Women's Hospital)
Cited by 89
Related Papers
Assessment of Resistance Mechanisms and Clinical Implications in Patients With<i>EGFR</i>T790M–Positive Lung Cancer and Acquired Resistance to Osimertinib
|JAMA Oncology|2018|689
Response of a Neuronal Model of Tuberous Sclerosis to Mammalian Target of Rapamycin (mTOR) Inhibitors: Effects on mTORC1 and Akt Signaling Lead to Improved Survival and Function
|Journal of Neuroscience|2008|502
Loss of Tsc1/Tsc2 activates mTOR and disrupts PI3K-Akt signaling through downregulation of PDGFR
|Journal of Clinical Investigation|2003|479
A Mouse Model of Tuberous Sclerosis: Neuronal Loss of Tsc1 Causes Dysplastic and Ectopic Neurons, Reduced Myelination, Seizure Activity, and Limited Survival
|Journal of Neuroscience|2007|460
Polycystin-2, the protein mutated in autosomal dominant polycystic kidney disease (ADPKD), is a Ca <sup>2+</sup> -permeable nonselective cation channel
|Proceedings of the National Academy of Sciences|2000|447