The combination of intravenous Reolysin and gemcitabine induces reovirus replication and endoplasmic reticular stress in a patient with KRAS-activated pancreatic cancer

Devalingam Mahalingam(The University of Texas at San Antonio Health Science Center), Steffan T. Nawrocki(University of Arizona), Sukeshi Patel Arora(The University of Texas MD Anderson Cancer Center), Giovanni Selvaggi, Gerard J. Nuovo(Life Sciences Discovery Fund), Matt Coffey(Oncolytics Biotech (Canada)), George M. Gill(Oncolytics Biotech (Canada))
BMC Cancer
July 9, 2015
10.1186/s12885-015-1518-0
Cited by 19

Related Papers

Survivin: Key Regulator of Mitosis and Apoptosis and Novel Target for Cancer Therapeutics
|Clinical Cancer Research|2008|782
Targeting autophagy augments the anticancer activity of the histone deacetylase inhibitor SAHA to overcome Bcr-Abl–mediated drug resistance
|Blood|2007|485
Histone deacetylase inhibitors: Mechanisms of cell death and promise in combination cancer therapy
|Cancer Letters|2008|426
Bortezomib Inhibits PKR-Like Endoplasmic Reticulum (ER) Kinase and Induces Apoptosis via ER Stress in Human Pancreatic Cancer Cells
|Cancer Research|2005|324
Targeting HSP90 for cancer therapy
|British Journal of Cancer|2009|320