The adaptive compensations in endocrine pancreas from glucocorticoid‐treated rats are reversible after the interruption of treatment
Alex Rafacho(Universidade Federal de Santa Catarina), José Roberto Bosqueiro(Universidade Estadual Paulista (Unesp)), Flávia M. Paula(Universidade Estadual de Campinas (UNICAMP)), Sebastião Roberto Taboga(Centro Universitário de Rio Preto), Antônio C. Boschero(Universidade Estadual de Campinas (UNICAMP)), Daniele Lisboa Ribeiro(Universidade Federal de Uberlândia), S. Quallio(Universidade Estadual de Campinas (UNICAMP))
Cited by 48
Related Papers
Glucocorticoid treatment and endocrine pancreas function: implications for glucose homeostasis, insulin resistance and diabetes
|Journal of Endocrinology|2014|214
A role for the putative cannabinoid receptor GPR55 in the islets of Langerhans
|Journal of Endocrinology|2011|119
Participation of prolactin receptors and phosphatidylinositol 3-kinase and MAP kinase pathways in the increase in pancreatic islet mass and sensitivity to glucose during pregnancy
|Journal of Endocrinology|2004|111
High doses of dexamethasone induce increased β-cell proliferation in pancreatic rat islets
|American Journal of Physiology-Endocrinology and Metabolism|2009|106