MID1, mutated in Opitz syndrome, encodes an ubiquitin ligase that targets phosphatase 2A for degradation
Alexander Trockenbacher(Universität Innsbruck), Susann Schweiger(Max Planck Institute for Molecular Genetics), Rainer Schneider(Universität Innsbruck), Vanessa Suckow(Max Planck Institute for Molecular Genetics), John Foerster(University of Dundee), Hans‐Hilger Ropers(Max Planck Institute for Molecular Genetics), Jennifer Winter(Max Planck Institute for Molecular Genetics), Sybille Krauß(German Center for Neurodegenerative Diseases)
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