Protein kinase C (PKC) participates in acetaminophen hepatotoxicity through c-jun-N-terminal kinase (JNK)-dependent and -independent signaling pathways
Behnam Saberi(University of Southern California), Neil Kaplowitz(University of Southern California)
Cited by 87
Related Papers
Betaine decreases hyperhomocysteinemia, endoplasmic reticulum stress, and liver injury in alcohol-fed mice
|Gastroenterology|2003|533
Drug-Induced Liver Injury: Cascade of Events Leading to Cell Death, Apoptosis or Necrosis
|International Journal of Molecular Sciences|2017|290
c‐Jun N‐terminal kinase mediates mouse liver injury through a novel Sab (SH3BP5)‐dependent pathway leading to inactivation of intramitochondrial Src
|Hepatology|2016|186
JNK interaction with Sab mediates ER stress induced inhibition of mitochondrial respiration and cell death
|Cell Death and Disease|2014|172