Suppression of monocyte function and differential regulation of IL-1 and IL-1ra by IL-4 contribute to resolution of experimental arthritis.
J B Allen(University of Vermont), Sharon M. Wahl(National Institutes of Health), Michał Bieńkowski(Gdańsk Medical University), H.L.S. Wong(University of Minnesota), G L Costa(National Institutes of Health)
Cited by 169
Related Papers
Suppression of arthritis by an inhibitor of nitric oxide synthase.
|The Journal of Experimental Medicine|1993|652
Surface proteins from Helicobacter pylori exhibit chemotactic activity for human leukocytes and are present in gastric mucosa.
|The Journal of Experimental Medicine|1992|325
Systemic and Local Interleukin-17 and Linked Cytokines Associated with Sjögren’s Syndrome Immunopathogenesis
|American Journal Of Pathology|2009|304
Macrophage- and astrocyte-derived transforming growth factor beta as a mediator of central nervous system dysfunction in acquired immune deficiency syndrome.
|The Journal of Experimental Medicine|1991|293
Rapid onset synovial inflammation and hyperplasia induced by transforming growth factor beta.
|The Journal of Experimental Medicine|1990|263