Differential PI3K signal transduction in obesity‐associated cardiac hypertrophy and response to ischemia
Bernhard Unsöld(University of Göttingen), Katrin Schäfer(University of Maryland, Baltimore)
Cited by 18
Related Papers
Endothelial p53 Deletion Improves Angiogenesis and Prevents Cardiac Fibrosis and Heart Failure Induced by Pressure Overload in Mice
|Journal of the American Heart Association|2015|141
Leptin promotes the mobilization of vascular progenitor cells and neovascularization by NOX2-mediated activation of MMP9
|Cardiovascular Research|2011|48
Importance of leptin signaling and signal transducer and activator of transcription-3 activation in mediating the cardiac hypertrophy associated with obesity
|Journal of Translational Medicine|2013|48
The endothelial tumor suppressor p53 is essential for venous thrombus formation in aged mice
|Blood Advances|2018|25
Arginase-1 Deletion in Erythrocytes Promotes Vascular Calcification via Enhanced GSNOR (S-Nitrosoglutathione Reductase) Expression and NO Signaling in Smooth Muscle Cells
|Arteriosclerosis Thrombosis and Vascular Biology|2022|18