Mitochondria in the etiology and pathogenesis of parkinson's disease
Anthony H.V. Schapira(Queen Mary University of London), Jonathan M. Cooper(University of Glasgow), Muxin Gu(Wellcome/MRC Cambridge Stem Cell Institute), Sarah J. Tabrizi(National Hospital for Neurology and Neurosurgery), Jan‐Willem Taanman(University College London), Michael W. J. Cleeter(University College London), Taylor A Seaton(The Royal Free Hospital)
Cited by 238
Related Papers
Huntington disease
|Nature Reviews Disease Primers|2015|1.6k
Mitofusin 1 and mitofusin 2 are ubiquitinated in a PINK1/parkin-dependent manner upon induction of mitophagy
|Human Molecular Genetics|2010|896
Mitochondrial defect in Huntington's disease caudate nucleus
|Annals of Neurology|1996|738
Priorities in Parkinson's disease research
|Nature Reviews Drug Discovery|2011|430
METTL1-mediated m7G modification of Arg-TCT tRNA drives oncogenic transformation
|Molecular Cell|2021|359