Reversal of <scp>SIN</scp>‐1‐induced e<scp>NOS</scp> dysfunction by the spin trap, <scp>DMPO</scp>, in bovine aortic endothelial cells via e<scp>NOS</scp> phosphorylation

Amlan Das(National Institute of Biomedical Genomics), Frederick A. Villamena(Lung Institute)
British Journal of Pharmacology
January 10, 2014
10.1111/bph.12572
Cited by 18

Related Papers

Deficiency of CCN5/WISP-2-Driven Program in breast cancer Promotes Cancer Epithelial cells to mesenchymal stem cells and Breast Cancer growth
|Scientific Reports|2017|31
CCN5 Activation by free or encapsulated-EGCG is required to Render Triple-negative breast cancer cell Viability and Tumor Progression
|Unknown|2022|20