Inducible Nitric Oxide Synthase and Inflammatory Diseases

Rubén Zamora(University of Pittsburgh), Timothy R. Billiar(University of Pittsburgh)

Molecular Medicine

May 1, 2000

10.1007/bf03401781

Cited by 543

Related Papers

Hepatic Ischemia/Reperfusion Injury Involves Functional TLR4 Signaling in Nonparenchymal Cells

|The Journal of Immunology|2005|361

Lipopolysaccharide Clearance, Bacterial Clearance, and Systemic Inflammatory Responses Are Regulated by Cell Type–Specific Functions of TLR4 during Sepsis

|The Journal of Immunology|2013|188

Central Role for MCP-1/CCL2 in Injury-Induced Inflammation Revealed by In Vitro, In Silico, and Clinical Studies

|PLoS ONE|2013|94

Endotoxin uptake in mouse liver is blocked by endotoxin pretreatment through a suppressor of cytokine signaling-1–dependent mechanism #

|Hepatology|2009|90

A Role for cGMP in Inducible Nitric-oxide Synthase (iNOS)-induced Tumor Necrosis Factor (TNF) α-converting Enzyme (TACE/ADAM17) Activation, Translocation, and TNF Receptor 1 (TNFR1) Shedding in Hepatocytes

|Journal of Biological Chemistry|2012|49