Integrated genomic-metabolic classification of acute myeloid leukemia defines a subgroup with NPM1 and cohesin/DNA damage mutationsGiorgia Simonetti, Giovanni Martinelli, Michela Tebaldi et al.|Leukemia|2021Cited by 37
Adult B-Cell Precursor Acute Lymphoblastic Leukemia (BC-ALL) Negative For Recurrent Fusion Genes Are Characterized By a High Complex Genetic Heterogeneity Influencing PrognosisIlaria Iacobucci, Giovanni Martinelli, Anna Ferrari et al.|Blood|2013Cited by 3
Alterations in Pathways Regulating Phosphatidil Inositol 3 Phosphate (PI3P) Produce Both Cell Proliferation and Therapy Resistance, and Define a Group of Patients with Poor Prognosis in Acute Myeloid Leukemia (AML)Giovanni Marconi, Giovanni Martinelli, Cristina Papayannidis et al.|Blood|2016Cited by 1
DEREGULATED EXPRESSION OF CHECKPOINT KINASE 1 (CHK1) IN BCR-ABL-POSITIVE ACUTE LYMPHOBLASTIC LEUKEMIA (BCR-ABL+ ALL) SUGGESTS A NEW THERAPEUTICTARGETAndrea Ghelli Luserna di Rorà, G. Martinelli, Ilaria Iacobucci et al.|Unknown|2013Cited by 0
Abstract 1766: Distinct pattern of alterations in tp53 mutated and wild type acute myeloid leukemia (AML) patientsAnna Ferrari, Martinelli Giovanni, Eugenio Fonzi et al.|Cancer Research|2017Cited by 0