Early Phenotypic Changes in Transgenic Mice That Overexpress Different Mutants of Amyloid Precursor Protein in BrainDieder Moechars, Fred Van Leuven, Ilse Dewachter et al.|Journal of Biological Chemistry|1999Cited by 668
A disintegrin-metalloproteinase prevents amyloid plaque formation and hippocampal defects in an Alzheimer disease mouse modelRolf Postina, Falk Fahrenholz, J. Bohl et al.|Journal of Clinical Investigation|2004Cited by 622
Neuronal Deficiency of Presenilin 1 Inhibits Amyloid Plaque Formation and Corrects Hippocampal Long-Term Potentiation But Not a Cognitive Defect of Amyloid Precursor Protein [V717I] Transgenic MiceIlse Dewachter, Fred Van Leuven, Delphine Reversé et al.|Journal of Neuroscience|2002Cited by 239
Mutant Presenilins Disturb Neuronal Calcium Homeostasis in the Brain of Transgenic Mice, Decreasing the Threshold for Excitotoxicity and Facilitating Long-term PotentiationIlka Schneider, Jochen Herms, Delphine Reversé et al.|Journal of Biological Chemistry|2001Cited by 136
A disintegrin-metalloproteinase prevents amyloid plaque formation and hippocampal defects in an Alzheimer disease mouse modelRolf Postina, Falk Fahrenholz, Anja Schroeder et al.|Journal of Clinical Investigation|2004Cited by 54