The Addiction-Susceptibility TaqIA/Ankk1 Controls Reward and Metabolism Through D2 Receptor-Expressing NeuronsEnrica Montalban, Serge Luquet, Giuseppe Gangarossa et al.|Biological Psychiatry|2023Cited by 21
CBP-HSF2 structural and functional interplay in Rubinstein-Taybi neurodevelopmental disorderAurélie de Thonel, Valérie Mezger, Johanna K. Ahlskog et al.|Nature Communications|2022Cited by 20
The addiction-susceptibility TaqIA/Ankyrin repeat and kinase domain containing 1 kinase (ANKK1) controls reward and metabolism through dopamine receptor type 2 (DR2)-expressing neuronsEnrica Montalban, Serge Luquet, Roman Walle et al.|bioRxiv (Cold Spring Harbor Laboratory)|2022Cited by 2
CBP/EP300-dependent acetylation and stabilization of HSF2 are compromised in the rare disorder, Rubinstein-Taybi syndromeAurélie de Thonel, Valérie Mezger, Johanna K. Ahlskog et al.|bioRxiv (Cold Spring Harbor Laboratory)|2018Cited by 2
CBP/EP300-dependent acetylation and stabilization of HSF2 are compromised in Rubinstein-Taybi syndromeAurélie de Thonel, Valérie Mezger, Johanna K. Ahlskog et al.|HAL (Le Centre pour la Communication Scientifique Directe)|2019Cited by 0