Genome-wide association study of clinically defined gout identifies multiple risk loci and its association with clinical subtypesHirotaka Matsuo, Nariyoshi Shinomiya, Atsushi Takahashi et al.|Annals of the Rheumatic Diseases|2015Cited by 183
ABCG2 dysfunction causes hyperuricemia due to both renal urate underexcretion and renal urate overloadHirotaka Matsuo, Nariyoshi Shinomiya, Akiyoshi Nakayama et al.|Scientific Reports|2014Cited by 163
Common dysfunctional variants of ABCG2 have stronger impact on hyperuricemia progression than typical environmental risk factorsAkiyoshi Nakayama, Nariyoshi Shinomiya, Hirotaka Matsuo et al.|Scientific Reports|2014Cited by 96