HIV-1 Escape from the CCR5 Antagonist Maraviroc Associated with an Altered and Less-Efficient Mechanism of gp120-CCR5 Engagement That Attenuates Macrophage TropismMichael Roche, Paul R. Gorry, Filippo Posta et al.|Journal of Virology|2011Cited by 82
HIV-1 predisposed to acquiring resistance to maraviroc (MVC) and other CCR5 antagonists in vitro has an inherent, low-level ability to utilize MVC-bound CCR5 for entryMichael Roche, Paul R. Gorry, Martin R. Jakobsen et al.|Retrovirology|2011Cited by 42